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Peer-Reviewed Study On COVID Vaccine Shows What Happened to 1 In 35 People

In recent years, any questions or criticism surrounding the COVID vaccine are effectively censored, banned, and labeled “false” by the almighty truth-bearers on social media.

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After undergoing a full peer review, a newly published study in the European Journal of Heart Failure found that 1 in 35 people showed signs of heart damage after taking the Moderna COVID-19 vaccine.

The CDC openly admits that myocarditis and pericarditis (inflammation of the outer lining of the heart) are “rare side effects of COVID vaccination, especially prevalent in adolescents and young men.” Despite this serious concern, the CDC says the vaccine is safe for “the vast majority of people,” and that data shows “the low risk of complications from the vaccine is outweighed by the reward.”

The study concluded that adverse effects were more common than previously thought. The study also found that adverse effects were more frequent in women versus men.

“One in 35 health care workers at a Swiss hospital had signs of heart injury associated with the vaccine, mRNA-1273, researchers found,” according to the Epoch Times.

Researchers wrote in the paper, “mRNA-1273 booster vaccination-associated elevation of markers of myocardial injury occurred in about one out of 35 persons (2.8%), a greater incidence than estimated in meta-analyses of hospitalized cases with myocarditis (estimated incidence 0.0035%) after the second vaccination.”

“In a generally healthy population, the level would be about 1 percent, the researchers said,” the report found.

Adverse effects continued for a period of 30 days and half of the individuals showed unusually high levels of high-sensitivity cardiac troponin T, which is an indicator of subclinical heart damage.

The long-term implications of the study remain unclear as little research has tracked people over time with heart injury.

“None of the patients experienced a major adverse cardiac event, such as heart failure, within 30 days of booster vaccination, and none had electrocardiogram changes,” the report added.

The possible side effects of the COVID-19 vaccine have been a topic of controversy in recent weeks. Business mogul and Tesla boss, Elon Musk, responded to the heart attack of LeBron James’ son, Bronny, by questioning the role played by the experimental and controversial COVID-19 vaccine.

Musk responded bluntly, “We cannot ascribe everything to the vaccine, but, by the same token, we cannot ascribe nothing. Myocarditis is a known side-effect. The only question is whether it is rare or common.”

In recent years, Americans have witnessed an alarming increase in young and healthy people suffering cardiac arrest. The 18-year-old son of NBA superstar LeBron James was rushed to a hospital after collapsing during a basketball workout.

Social media erupted with widespread speculation about what caused Bronny’s heart to stop despite being a young and healthy athlete. In recent years, any questions or criticism surrounding the COVID vaccine are effectively censored, banned, and labeled “false” by the almighty truth-bearers on social media.

Multi-billion dollar pharmaceutical companies are also wielding their power, influence, and money to shape the national discourse surrounding their drugs. Twitter boss Elon Musk recently exposed internal documents proving Pfizer board member Dr. Scott Gottlieb helped censor Americans online.

“Nothing to see, no questions to ask,” Charlie Kirk sarcastically wrote.

Bronny is entering his first year at USC and he is ranked as a top-25 recruit and future NBA prospect by ESPN. A James family spokesperson said, “Yesterday while practicing Bronny James suffered a cardiac arrest. Medical staff was able to treat Bronny and take him to the hospital.” Bronny was discharged from the hospital after making a full recovery.


Here is the study. I’ll print the abstract and link to the article itself.

Sex-specific differences in myocardial injury incidence after COVID-19 mRNA-1273 booster vaccination

Natacha Buergin, Pedro Lopez-Ayala Contributed equally as co-first authors.

Abstract

Aims

To explore the incidence and potential mechanisms of oligosymptomatic myocardial injury following COVID-19 mRNA booster vaccination.

Methods and results

Hospital employees scheduled to undergo mRNA-1273 booster vaccination were assessed for mRNA-1273 vaccination-associated myocardial injury, defined as acute dynamic increase in high-sensitivity cardiac troponin T (hs-cTnT) concentration above the sex-specific upper limit of normal on day 3 (48–96 h) after vaccination without evidence of an alternative cause. To explore possible mechanisms, antibodies against interleukin-1 receptor antagonist (IL-1RA), the SARS-CoV-2-nucleoprotein (NP) and -spike (S1) proteins and an array of 14 inflammatory cytokines were quantified. Among 777 participants (median age 37 years, 69.5% women), 40 participants (5.1%; 95% confidence interval [CI] 3.7–7.0%) had elevated hs-cTnT concentration on day 3 and mRNA-1273 vaccine-associated myocardial injury was adjudicated in 22 participants (2.8% [95% CI 1.7–4.3%]). Twenty cases occurred in women (3.7% [95% CI 2.3–5.7%]), two in men (0.8% [95% CI 0.1–3.0%]). Hs-cTnT elevations were mild and only temporary. No patient had electrocardiographic changes, and none developed major adverse cardiac events within 30 days (0% [95% CI 0–0.4%]). In the overall booster cohort, hs-cTnT concentrations (day 3; median 5, interquartile range [IQR] 4–6 ng/L) were significantly higher compared to matched controls (n = 777, median 3 [IQR 3–5] ng/L, p < 0.001). Cases had comparable systemic reactogenicity, concentrations of anti-IL-1RA, anti-NP, anti-S1, and markers quantifying systemic inflammation, but lower concentrations of interferon (IFN)-λ1 (IL-29) and granulocyte-macrophage colony-stimulating factor (GM-CSF) versus persons without vaccine-associated myocardial injury.

Conclusion

mRNA-1273 vaccine-associated myocardial injury was more common than previously thought, being mild and transient, and more frequent in women versus men. The possible protective role of IFN-λ1 (IL-29) and GM-CSF warrant further studies.

Find the paper here

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